By F.J. Dixon, Henry G. Kunkel (Eds.)
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Extra info for Advances in Immunology, Vol. 18
1966). The offspring of this mouse were used to initiate a selection experiment in which male and feniale nonresponders to BSA were matcd to produce the next generation. , 1966). Subsequent experiments showed that with low doses of BSA, the putative nonresponder or Sobey mice were as reactive as responder Swiss white mice. However, when antigen doscs greater than 500 pg. were injected, Sobey mice frequently became tolerant whereas Swiss white mice did not (Hardy and Rowley, 1968). The genes for unresponsiveness to BSA, therefore, appear to be involved in lowering the threshold of tolerancc induction.
In some rabbits this enzyme behaved like a hapten-carrier system in that the haptenic B subunits induced antibody formation only if combined with carrier A subunits. In other rabbits the B subunits alone were immunogenic, and the ability to respond to B, was inherited as a genetic trait (Rajewsky and RottlBnder, 1967). Hyporesponsiveness to the A,B, enzyme could be induced by injection of LDH-A, into newborn rabbits. Both anti-A and anti-B titers were equally depressed. The response to A,B, was not depressed by neonatal injections of LDH-B,.
The 1R kind 2R chroinosonies are conil)ined as the H - 2 products have not yet been distinguished; this is also true of 3 R and 5R. , 197211. , 197213). The results of testing the various recombinants are summarized in Fig.